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Its enhanced stability and high receptor affinity produce consistent activation of downstream pathwaysmTOR, MAPK, and PI3K/Aktcritical for studies of cell proliferation , protein synthesis , and glucose metabolism

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John Oldham, a bariatric surgeon with Baptist Health

If we extend this observation to adult neurogenesis, promoter hypermethylation might not be responsible for the final repression of intrinsic fate-determination TFs, which occurs in later stages of neuronal development (Figure 1)

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